New study demonstrates that one type of lipid is so critical for immune evasion that certain cancers cannot proliferate without them. Modulating intake of dietary lipids could open up an avenue for treatment.
The study explains why treatments for many autoimmune and inflammatory diseases raise the risk of contracting TB—and also upends long-held assumptions about what kicks off an immune response.
A Rockefeller employee since 2001, Chin will be missed by many in the community who came to rely on his deep technical expertise and graceful approach to problem solving.
Hyperactivity, repetitive behavior, and language deficits all appeared in the mice, highlighting an unexpected role the cerebellum seems to play in autism.
Researchers found a “neuro-cancer axis” that may promote the spread of breast cancer. They also found that an FDA-approved anti-nausea medication may prevent it.
Students in the BIOGROW program participate in professional development and community building events in collaboration with the university's Office of Diversity, Equity, and Inclusion.
Victora, who studies how antibodies are generated in response to infection and immunization, is among 26 scientists nationwide to receive this designation.
Four community members—Sadye Paez, Elizabeth Campbell, Yuriria Vázquez, and Chad Morton—were recognized for their advocacy work both within the university and in the wider scientific community.
A novel platform for identifying metabolic gene functions has already revealed interactions between proteins and metabolites that are fundamental to cell metabolism.
Vosshall is recognized for her pioneering studies of the mosquito Aedes aegypti, which transmits pathogens causing human diseases including dengue, Zika, chikungunya, and yellow fever.
With a new microscope that's as light as a penny, researchers can now observe broad swaths of the brain in action as mice move about and interact with their environments.
Since first detected in birds in 2021, avian flu has killed millions of poultry and infected animals once thought to be immune. What early warning signs could point to an increasing risk for humans?
New findings describe how the enzyme CST is recruited to the end of the telomere, where it maintains telomere length with the help of subtle chemical changes made to the protein POT1.
The advent of AlphaFold3 could be an unprecedented boon for drug development. Computational biologist Jiankun Lyu discusses the pros and cons of the tech, and his work analyzing the algorithm.
Bieniasz is being honored for his work on the life cycle of retroviruses and their interactions with host proteins. With his election, nearly half of Rockefeller faculty are NAS members.
The findings in mice may explain how addictive drugs hijack natural reward processing systems, creating the urge to use while throwing natural urges to eat and drink off-kilter.
Thomas Tuschl has devoted his career to making discoveries that bridge the gap between bench and business—and have resulted in entirely new classes of drugs.
The author of There Are Places in the World Where Rules Are Less Important Than Kindness was presented with Rockefeller’s prestigious science writing award on April 9.
SNFiRU, which aims to better understand the agents that cause infectious disease and to lower barriers to treatment and prevention globally, launched numerous initiatives in its inaugural year.
Retinoic acid, the active state of Vitamin A, appears to regulate how stem cells enter and exit a transient state central to their role in wound repair.
uLIPSTIC, an updated method for directly observing physical interactions between cells, could allow scientists to one day map every possible cell interaction.
A novel platform allows researchers to directly target enzymes implicated in infectious diseases, several genetic disorders, and some cancers for the first time.
Mojsov's research into hormonal triggers for insulin production led to groundbreaking treatments for diabetes and obesity. She will be presented with the award on April 17.
The end replication problem dictates that telomeres shrink unless telomerase intervenes. But the problem is actually twice as complicated, with telomerase providing only part of the solution.
The absence of a single immune cell receptor has been linked to both fewer defenses against mycobacterial infections, such as TB, and damaging buildup of sticky residue in the lungs.
