Vosshall is recognized for her pioneering studies of the mosquito Aedes aegypti, which transmits pathogens causing human diseases including dengue, Zika, chikungunya, and yellow fever.
With a new microscope that's as light as a penny, researchers can now observe broad swaths of the brain in action as mice move about and interact with their environments.
Since first detected in birds in 2021, avian flu has killed millions of poultry and infected animals once thought to be immune. What early warning signs could point to an increasing risk for humans?
New findings describe how the enzyme CST is recruited to the end of the telomere, where it maintains telomere length with the help of subtle chemical changes made to the protein POT1.
The advent of AlphaFold3 could be an unprecedented boon for drug development. Computational biologist Jiankun Lyu discusses the pros and cons of the tech, and his work analyzing the algorithm.
Bieniasz is being honored for his work on the life cycle of retroviruses and their interactions with host proteins. With his election, nearly half of Rockefeller faculty are NAS members.
The findings in mice may explain how addictive drugs hijack natural reward processing systems, creating the urge to use while throwing natural urges to eat and drink off-kilter.
Thomas Tuschl has devoted his career to making discoveries that bridge the gap between bench and business—and have resulted in entirely new classes of drugs.
The author of There Are Places in the World Where Rules Are Less Important Than Kindness was presented with Rockefeller’s prestigious science writing award on April 9.
SNFiRU, which aims to better understand the agents that cause infectious disease and to lower barriers to treatment and prevention globally, launched numerous initiatives in its inaugural year.
Retinoic acid, the active state of Vitamin A, appears to regulate how stem cells enter and exit a transient state central to their role in wound repair.
uLIPSTIC, an updated method for directly observing physical interactions between cells, could allow scientists to one day map every possible cell interaction.
A novel platform allows researchers to directly target enzymes implicated in infectious diseases, several genetic disorders, and some cancers for the first time.
Mojsov's research into hormonal triggers for insulin production led to groundbreaking treatments for diabetes and obesity. She will be presented with the award on April 17.
The end replication problem dictates that telomeres shrink unless telomerase intervenes. But the problem is actually twice as complicated, with telomerase providing only part of the solution.
The absence of a single immune cell receptor has been linked to both fewer defenses against mycobacterial infections, such as TB, and damaging buildup of sticky residue in the lungs.
A class of ineffective immune cells may be driving Alzheimer’s disease, a finding that could both explain why APOE4 gene increases the risk of the disease and why a new drug can impact it.
Luciano Marraffini’s research helped lay the groundwork for the newly FDA-approved CRISPR-based therapy for sickle cell anemia. He reflects on how we got here—and where the science is going next.
By focusing on the emergent features of cell collectives, instead of individual cells, scientists forge a new path for understanding how organs develop their architecture.
From plumbing the depths of wound repair to tackling how songbirds solve problems, here are some fascinating discoveries that came out of Rockefeller in 2023.
Carlo Yuvienco, the inaugural director of the university’s new Ford Center Incubator, on how the new space will help translate research breakthroughs into market-ready biomedical innovations.
Mojsov’s research directly led to blockbuster drugs like Ozempic and Wegovy. She talks about the long quest for proper credit and lessons that can be learned from her experience.
“I believe this is going to be a very fruitful find. Every time people have studied nutrient sensing, we’ve learned a lot about biology, and many drugs have been developed as a result.”
Why do our bodies not only fail to learn from prior dengue infection but also become more vulnerable to it as a result? New research pinpoints a subgroup of antibodies that may be to blame.
