New findings describe how the enzyme CST is recruited to the end of the telomere, where it maintains telomere length with the help of subtle chemical changes made to the protein POT1.
The advent of AlphaFold3 could be an unprecedented boon for drug development. Computational biologist Jiankun Lyu discusses the pros and cons of the tech, and his work analyzing the algorithm.
The findings in mice may explain how addictive drugs hijack natural reward processing systems, creating the urge to use while throwing natural urges to eat and drink off-kilter.