News & HighlightsNews
Cancer cells may be using lipids to hide from the immune system
New study demonstrates that one type of lipid is so critical for immune evasion that certain cancers cannot proliferate without them. Modulating intake of dietary lipids could open up an avenue for treatment.
New findings on TB could change how we treat inflammatory disorders    
The study explains why treatments for many autoimmune and inflammatory diseases raise the risk of contracting TB—and also upends long-held assumptions about what kicks off an immune response.      
This technique for studying cell receptors could have sweeping implications for drug development
Researchers created a tool capable of comprehensively mapping crucial interactions underlying drug efficacy in one superfamily of cell receptors.
New insight into the protein mutations that cause Rett syndrome
Findings bolster the idea that the functions of this protein—MeCP2—are more centered on nucleosomes, rather than other forms of DNA.
Second genetic sensor for DNA methylation discovered
CDCA7, whose mutations alter DNA methylation pattern and cause immunodeficiency, is a novel sensor for a special class of methylated DNA.
Surprising mechanism for removing dead cells identified
A tandem signaling process turns ordinary cells into an efficient cleanup crew.
Knocking out one key gene leads to autistic traits
Hyperactivity, repetitive behavior, and language deficits all appeared in the mice, highlighting an unexpected role the cerebellum seems to play in autism.
Link discovered between sensory neurons and breast cancer metastasis
Researchers found a “neuro-cancer axis”  that may promote the spread of breast cancer. They also found that an FDA-approved anti-nausea medication may prevent it.
New genetic tool could identify drug targets for diseases associated with metabolic dysfunction
A novel platform for identifying metabolic gene functions has already revealed interactions between proteins and metabolites that are fundamental to cell metabolism.
Asexual reproduction usually leads to a lack of genetic diversity. Not for these ants.
Parthenogenic species must compensate for their limited gene pool or risk extinction.
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