The study is the first to identify a damage response pathway that is distinct from but parallel to the classical pathway triggered by pathogens.

Damaged gums may release bacteria into the bloodstream that trigger arthritis flare-ups, potentially explaining why people with gum disease don't respond as well to arthritis treatments.

Just as the Rice lab’s work on HCV exposed that virus’s weaknesses, the hope is that this novel approach could do the same for HBV.

The anterior thalamus plays such a key role in memory that boosting it in mice consolidates the animals' trivial experiences into long-term memories.

The tech, dubbed MesoLF, captures 10,500 neurons buried at once-inaccessible depths, firing from brain regions many millimeters apart, simultaneously—all with unprecedented resolution.

The relatively simple and low-cost procedure could empower laboratories in low-resource areas to generate nanobodies against SARS-CoV-2, as well as other viruses.

The findings offer a new way to understand how some ants become total layabouts.

A trio of faulty genes fail to put the brakes on the immune system’s all-out assault on SARS-CoV-2, leading to the inflammatory overload characteristic of MIS-C.

Starving cancer cells of a key amino acid could potentially render tumors more vulnerable to the body’s natural immune response.

The body's first blush with a pathogen shapes how it will respond to vaccines. New evidence clarifies how this phenomenon works, mechanistically.