Calendar of Events & Lectures

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Tuesday, September 16, 2025

Pre-Seminar Coffee and Cookies

| LOWER LEVEL GREENBERG BUILDING (CRC)
Seminars, Center for Studies in Physics and Biology Seminars

AI for Autonomous Agents: Sequence AI and Peer Cooperative Lifelong Learning.

| CARSON FAMILY AUDITORIUM (CRC)
Seminars, Center for Studies in Physics and Biology Seminars
Hava Siegelmann, Ph.D., Provost Professor, University of Massachusetts, Amherst

2025 Pearl Meister Greengard Prize Reception and Ceremony

| CASPARY AUDITORIUM
Special Events, Pearl Meister Greengard Prize
Reception 5:45 PM l Ceremony 6:30 PM
Maria Jasin, Ph.D., William E. Snee Chair, Developmental Biology Program, MSKCC

Tri-Institutional Calendars

The close proximity among the three institutions which comprise the Tri-I has led to a culture that encourages interinstitutional interactions and shared resources, including access to lectures and seminars from internationally renowned scientists and clinicians:

Upcoming Events

From Ancestry to Biochemistry in Steatotic Liver Disease

| CASPARY AUDITORIUM
The Joshua Lederberg Distinguished Lecture in Molecular Genetics

Self-Organization and Symmetry Breaking in Human Embryo and Models

| CARSON FAMILY AUDITORIUM (CRC)

Rare Sugar Probes to Identify Glycan Binding Proteins

| 116 ROCKEFELLER RESEARCH LABORATORIES, MSKCC, 430 E. 67TH ST.

News

Why understanding stem cells lies as the root of treating diseases from psoriasis to cancer

Elaine Fuchs has spent decades uncovering why our bodies are so good at regenerating skin—and how we might harness that understanding to combat illness, hair loss, and perhaps even the aging process itself.

New model demonstrates how a dynamic mechanism regulates traffic through the nuclear pore complex

A new study reveals that nuclear pore complexes—tiny gateways in a cell’s nuclear membrane—are governed by dynamic action.

Scientists can finally answer an old question about cellular aging

Replicative aging of human cells, the result of telomere shortening, is slower at physiological oxygen than at atmospheric oxygen, a difference now shown to be due to low oxygen impairing the ATM kinase response to withered telomeres.

Room Reservation Calendar